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PharmaGABA Chewables 200 mg 60 tabs by Designs for Health

0.32 LBS
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Product Description

PharmaGABA Chewables 200 mg 60 tabs

Dietary Supplement

PharmaGABA™ is a proprietary form of GABA, one of the body’s main calming neurotransmitters, manufactured through a natural fermentation process which has
been shown to result in a more clinically effective GABA than generic synthetic forms.

Does not contain gluten.

Recommended Use: As a dietary supplement, chew two tablets per day, or as directed by your health care practitioner.

Supplement Facts
Serving Size 2 tablets
Servings Per Container 30
Amount Per Serving
gamma-Aminobutyric acid (as PharmaGABA™) 200 mg

Other Ingredients: Xylitol, F-MELT®, vegetable stearates, microcystalline cellulose, citric acid, natural flavors, stevia (leaf) extract, pharmaceutical


PharmaGABA™ is a trademark of Pharma Foods International Co., Ltd.
F-MELT® is a registered trademark of Fuji Chemical Industry Co., Ltd.
Notice: Color, size or shape may appear diferent between lots.

* These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure, or prevent any disease.

Product Videos

2-Minute Neuroscience: GABA (01:59)
In this video I discuss the neurotransmitter gamma-aminobutyric acid, or GABA. GABA is the primary inhibitory neurotransmitter in the human nervous system; its effects generally involve making neurons less likely to fire action potentials or release neurotransmitters. GABA acts at both ionotropic (GABAa) and metabotropic (GABAb) receptors, and its action is terminated by a transporter called the GABA transporter. Several drugs like alcohol and benzodiazepines cause increased GABA activity, which is associated with sedative effects. TRANSCRIPT: Welcome to 2 minute neuroscience, where I simplistically explain neuroscience topics in 2 minutes or less. In this installment I will discuss gamma-aminobutyric acid, or GABA. Although GABA’s primary functions are as a neurotransmitter, it has the structure of an amino acid and thus is referred to as an amino acid neurotransmitter. It is synthesized from another amino acid neurotransmitter, glutamate, in a reaction catalyzed by the enzyme glutamic acid decarboxylase. The function of GABA changes over the course of neural development, but in the mature brain it acts primarily as an inhibitory neurotransmitter; in other words when GABA interacts with the receptors of a neuron, it generally makes the neuron less likely to fire an action potential or release neurotransmitters. There are two types of receptors GABA interacts with, GABAa and GABAb receptors. GABAa receptors are ionotropic receptors. When GABA binds to the GABAa receptor, it causes the opening of an associated ion channel that is permeable to the negatively charged ion chlorine. When negative chlorine ions flow into the neuron, they hyperpolarize the membrane potential of the neuron and make it less likely the neuron will fire an action potential. GABAb receptors are metabotropic (or g-protein coupled) receptors; when activated they frequently cause the opening of potassium channels. These channels allow positively charged potassium ions to flow out of the neuron, again making the neuron hyperpolarized and less likely to fire an action potential. The actions of GABA are terminated by proteins called GABA transporters, which transport GABA from the synaptic cleft into neurons or glial cells where it is degraded primarily by mitochondrial enzymes. Because GABA can reduce neural transmission, increased GABA activity can have sedative effects. Accordingly, a number of drugs that have such effects, like alcohol and benzodiazepines, increase activity at the GABA receptor.
  • 2-Minute Neuroscience: GABA
    In this video I discuss the neurotransmitter gamma-aminobutyri...
  • GABA Neurotransmitters, Anxiety, and the Dangers of Benzodiazepines
    Dr. Von Stieff explains the dangers of what benzodiazepines do...

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